Bisfenol A (Polski)

Brotons, J.A., Olea-Serrano, M.F., Villalobos, M., Pedraza, V., Olea, N. „Xenoestrogens released from lacquer coatings in food cans.” Environmental Health Perspectives 103, 6 (1995): 608-12. doi:10.1289/ehp.95103608.

Lakind, Judy S, and Daniel Q Naiman. „Dzienne spożycie bisfenolu A i potencjalne źródła narażenia: 2005-2006 National Health and Nutrition Examination Survey”. Journal of Exposure Science & Environmental Epidemiology 21, 3 (2011): 272-9. doi:10.1038/jes.2010.9.

Vandenberg, Laura N et al. „Human exposure to bisphenol A (BPA).” Reproductive Toxicology 24, 2 (2007): 139-77. doi:10.1016/j.reprotox.2007.07.010.

Rochester, Johanna R, and Ashley L Bolden. „Bisphenol S and F: A Systematic Review and Comparison of the Hormonal Activity of Bisphenol A Substitutes.” Environmental Health Perspectives 123, 7 (2015): 643-50. doi:10.1289/ehp.1408989.

Ji, Kyunghee, Hong, Seongjin Hong, Younglim Kho and Kyungho Choi. „Effects of bisphenol S exposure on endocrine functions and reproduction of zebrafish.” Environmental Science & Technology 47,15 (2013): 8793-800. doi:10.1021/es400329t.

Kim, Ji-Youn et al. „Effects of bisphenol compounds on the growth and epithelial mesenchymal transition of MCF-7 CV human breast cancer cells.” Journal of Biomedical Research 31,4 (2017): 358-369. doi:10.7555/JBR.31.20160162.

Kalifornijskie Biuro Oceny Środowiskowych Zagrożeń Zdrowia. „Chemicals: Bisfenol-A.” Ostatnio zmieniony 2020. http://oehha.ca.gov/chemicals/bisphenol-a.

Xue, Jingchuan et al. „Bisphenols, Benzophenones, and Bisphenol A Diglycidyl Ethers in Textiles and Infant Clothing.” Environmental Science & Technology 51,9 (2017): 5279-5286. doi:10.1021/acs.est.7b00701.

Matsumoto, H, S. Adachi and Y. Suzuki. „Bisfenol A w cząstkach stałych powietrza atmosferycznego odpowiedzialny za proliferację komórek ludzkiego raka piersi MCF-7 i jego zmiany stężenia w ciągu 6 miesięcy”. Archives of Environmental Contamination and Toxicology 48,4 (2005): 459-66. doi:10.1007/s00244-003-0243-x.

Rudel Ruthann, David E. Camann, John D. Spengler, Leo R. Korn and Julia G Brody. „Ftalany, alkilofenole, pestycydy, polibromowane etery difenylowe i inne związki zaburzające gospodarkę hormonalną w powietrzu i kurzu w pomieszczeniach.” Environmental Science & Technology 37,20 (2003): 4543-53. doi:10.1021/es0264596.

Rodriguez-Mozaz S, ML de Alda and D. Barceló. „Analysis of bisphenol A in natural waters by means of an optical immunosensor”. Water Research 39 (2005): 5071-9. doi:10.1016/j.watres.2005.09.023.

Carwile, Jenny L et al. „Polycarbonate bottle use and urinary bisphenol A concentrations.” Environmental Health Perspectives 117, 9 (2009): 1368-72. doi:10.1289/ehp.0900604.

Smith R, Lourie. Slow Death by Rubber Duck: The Secret Danger of Everyday Things. Berkeley, CA: Counterpoint, 2009.

Rudel, Ruthann A et al. „Food packaging and bisphenol A and bis(2-ethyhexyl) phthalate exposure: findings from a dietary intervention.” Environmental Health Perspectives 119,7 (2011): 914-20. doi:10.1289/ehp.1003170.

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Stahlhut, Richard W, Wade V Welshons and Shanna H Swan. „Bisfenol A dane w NHANES sugerują dłuższy niż oczekiwano okres półtrwania, znaczną ekspozycję nieżywnościową lub oba.” Environmental Health Perspectives 117,5 (2009): 784-9. doi:10.1289/ehp.0800376.

vom Saal, Frederick S, and Wade V Welshons. „Evidence that bisphenol A (BPA) can be accurately measured without contamination in human serum and urine, and that BPA causes numerous hazards from multiple routes of exposure.” Molecular and Cellular Endocrinology 398,1-2 (2014): 101-13. doi:10.1016/j.mce.2014.09.028.

Rochester, Johanna R, and Ashley L Bolden. „Bisphenol S and F: A Systematic Review and Comparison of the Hormonal Activity of Bisphenol A Substitutes.” Environmental Health Perspectives 123,7 (2015): 643-50. doi:10.1289/ehp.1408989.

Kalifornijskie Biuro Oceny Zagrożeń Zdrowia Środowiskowego. „Chemicals Considered or Listed Under Proposition 65: Bisphenol A (BPA).” Ostatnio zmieniony 2020. http://oehha.ca.gov/proposition-65/chemicals/bisphenol-bpa.

Stahlhut, Richard W, Wade V Welshons i Shanna H Swan. „Bisfenol A dane w NHANES sugerują dłuższy niż oczekiwano okres półtrwania, znaczną ekspozycję nieżywnościową lub oba.” Environmental Health Perspectives 117,5 (2009): 784-9. doi:10.1289/ehp.0800376.

Ye, Xibiao et al. „Levels of metabolites of organophosphate pesticides, phthalates, and bisphenol A in pooled urine specimens from pregnant women participating in the Norwegian Mother and Child Cohort Study (MoBa).” International Journal of Hygiene and Environmental Health 212, 5 (2009): 481-91. doi:10.1016/j.ijheh.2009.03.004.

Padmanabhan, V et al. „Maternal bisfenol-A poziomy przy dostawie: looming problem?”. Journal of Perinatology: Official Journal of the California Perinatal Association 28,4 (2008): 258-63. doi:10.1038/sj.jp.7211913.

Teeguarden Justin G, Nathan C. Twaddle, Mona I. Churchwell and Daniel R. Doerge. „Urine and serum biomonitoring of exposure to environmental estrogens I: Bisphenol A in pregnant women”. Food and Chemical Toxicology 92 (2016): 129-142. https://doi.org/10.1016/j.fct.2016.03.023.

Kuruto-Niwa Ryoko et al. „Aktywność estrogenna chlorowanych pochodnych estrogenów i flawonoidów z wykorzystaniem systemu ekspresji GFP.” Environmental Toxicology and Pharmacology 23, 1 (2007): 121-128. https://doi.org/10.1016/j.etap.2006.07.011.

Sun, Yen et al. „Determination of bisphenol A in human breast milk by HPLC with column-switching and fluorescence detection.” Biomedical Chromatography: BMC 18,8 (2004): 501-7. doi:10.1002/bmc.345.

Ikezuki, Yumiko et al. „Determination of bisphenol A concentrations in human biological fluids reveals significant early prenatal exposure.” Human Reproduction 17,11 (2002): 2839-41. doi:10.1093/humrep/17.11.2839.

Environmental Working Group. „BPA i alternatywy.” Accessed November 4, 2020. https://www.ewg.org/key-issues/toxics/bpa.

Schönfelder, Gilbert et al. „Parent bisphenol A accumulation in the human maternal-fetal-placental unit.” Environmental Health Perspectives 110,11 (2002): A703-7. doi:10.1289/ehp.110-1241091.

Calafat, Antonia M et al. „Exposure to bisphenol A and other phenols in neonatal intensive care unit premature infants.” Environmental Health Perspectives 117,4 (2009): 639-44. doi:10.1289/ehp.0800265.

Vandenberg, Laura N et al. „Exposure to environmentally relevant doses of the xenoestrogen bisphenol-A alters development of the fetal mouse mammary gland.” Endocrinology 148,1 (2007): 116-27. doi:10.1210/en.2006-0561.

Vandenberg, Laura N et al. „Perinatal exposure to the xenoestrogen bisphenol-A induces mammary intraductal hyperplasias in adult CD-1 mice.” Reproductive Toxicology 26,3-4 (2008): 210-9. doi:10.1016/j.reprotox.2008.09.015.

Betancourt, Angela M et al. „Proteomic analysis in mammary glands of rat offspring exposed in utero to bisphenol A.” Journal of Proteomics 73,6 (2010): 1241-53. doi:10.1016/j.jprot.2010.02.020.

Perrot-Applanat, Martine et al. „Alteration of mammary gland development by bisphenol a and evidence of a mode of action mediated through endocrine disruption.” Molecular and Cellular Endocrinology 475 (2018): 29-53. doi:10.1016/j.mce.2018.06.015.

Markey, C M et al. „In utero exposure to bisphenol A alters the development and tissue organization of the mouse mammary gland.” Biology of Reproduction 65,4 (2001): 1215-23. doi:10.1093/biolreprod/65.4.1215.

Maffini, Maricel V et al. „Endocrine disruptors and reproductive health: the case of bisphenol-A.” Molecular and Cellular Endocrinology 254-255 (2006): 179-86. doi:10.1016/j.mce.2006.04.033.

Muñoz-de-Toro, Monica et al. „Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice.” Endocrinology 146,9 (2005): 4138-47. doi:10.1210/en.2005-0340.

Murray, Tessa J et al. „Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure.” Reproductive Toxicology 23,3 (2007): 383-90. doi:10.1016/j.reprotox.2006.10.002.

Acevedo, Nicole et al. „Perinatally administered bisphenol a as a potential mammary gland carcinogen in rats.” Environmental Health Perspectives 121,9 (2013): 1040-6. doi:10.1289/ehp.1306734.

Durando, Milena et al. „Prenatal bisphenol A exposure induces preneoplastic lesions in the mammary gland in Wistar rats.” Environmental Health Perspectives 115,1 (2007): 80-6. doi:10.1289/ehp.9282.

Jenkins Sarah et al. „Oral exposure to bisphenol a increases dimethylbenzanthracene-induced mammary cancer in rats.” Environmental Health Perspectives 117, 6 (2009): 910-15. doi:10.1289/ehp.11751.

Acevedo, Nicole et al. „Perinatally administered bisphenol a as a potential mammary gland carcinogen in rats.” Environmental Health Perspectives 121,9 (2013): 1040-6. doi:10.1289/ehp.1306734.

Wadia, Perinaaz R et al. „Low-dose BPA exposure alters the mesenchymal and epithelial transcriptomes of the mouse fetal mammary gland.” PloS One 8, 5 (2013): e63902. doi:10.1371/journal.pone.0063902.

Ibrahim, Marwa A A et al. „Effect of bisphenol A on morphology, apoptosis and proliferation in the resting mammary gland of the adult albino rat.” International Journal of Experimental Pathology 97, 1 (2016): 27-36. doi:10.1111/iep.12164.

Wadia, Perinaaz R et al. „Perinatal bisphenol A exposure increases estrogen sensitivity of the mammary gland in diverse mouse strains.” Environmental Health Perspectives 115, 4 (2007): 592-8. doi:10.1289/ehp.9640.

Ayyanan, Ayyakkannu et al. „Perinatal exposure to bisphenol a increases adult mammary gland progesterone response and cell number.” Molecular Endocrinology 25, 11 (2011): 1915-23. doi:10.1210/me.2011-1129.

Moral, Raquel et al. „Effect of prenatal exposure to the endocrine disruptor bisphenol A on mammary gland morphology and gene expression signature.” The Journal of Endocrinology 196,1 (2008): 101-12. doi:10.1677/JOE-07-0056.

Fischer, Catha et al. „Bisphenol A (BPA) Exposure In Utero Leads to Immunoregulatory Cytokine Dysregulation in the Mouse Mammary Gland: A Potential Mechanism Programming Breast Cancer Risk.” Hormones & Cancer 7,4 (2016): 241-51. doi:10.1007/s12672-016-0254-5.

Goodson, William H 3rd et al. „Activation of the mTOR pathway by low levels of xenoestrogens in breast epithelial cells from high-risk women.” Carcinogenesis 32, 11 (2011): 1724-33. doi:10.1093/carcin/bgr196.

Tilghman, Syreeta L et al. „Endocrine disruptor regulation of microRNA expression in breast carcinoma cells.” PloS One 7,3 (2012): e32754. doi:10.1371/journal.pone.0032754.

Weng, Yu-I et al. „Epigenetic influences of low-dose bisphenol A in primary human breast epithelial cells.” Toxicology and Applied Pharmacology 248, 2 (2010): 111-21. doi:10.1016/j.taap.2010.07.014.

Xu F et al. „Bisfenol A indukuje działanie proliferacyjne zarówno na komórki raka piersi, jak i komórki śródbłonka naczyniowego poprzez wspólną ścieżkę zależną od GPER w niedotlenieniu.” Environmental Pollution 231 (2017):1609-1620. doi: 10.1016/j.envpol.2017.09.069.

Dairkee, Shanaz H et al. „Bisphenol A indukuje profil agresywności nowotworowej w komórkach wysokiego ryzyka pochodzących od pacjentek z rakiem piersi.” Cancer Research 68, 7 (2008): 2076-80. doi:10.1158/0008-5472.CAN-07-6526.

Xu F et al. „Bisphenol A indukuje działanie proliferacyjne zarówno na komórki raka piersi, jak i komórki śródbłonka naczyniowego poprzez wspólną ścieżkę zależną od GPER w niedotlenieniu.” Environmental Pollution 231 (2017):1609-1620. doi: 10.1016/j.envpol.2017.09.069.

Lapensee, Elizabeth W et al. „Bisphenol A w niskich nanomolarnych dawkach confers chemoresistance in estrogen receptor-alpha-positive and -negative breast cancer cells.” Environmental Health Perspectives 117, 2 (2009): 175-80. doi:10.1289/ehp.11788.

LaPensee, Elizabeth W et al. „Bisphenol A and estradiol are equipotent in antagonizing cisplatin-induced cytotoxicity in breast cancer cells.” Cancer Letters 290, 2 (2010): 167-73. doi:10.1016/j.canlet.2009.09.005.

Tucker, Deirdre K et al. „Evaluation of Prenatal Exposure to Bisphenol Analogues on Development and Long-Term Health of the Mammary Gland in Female Mice.” Environmental Health Perspectives 126, 8 (2018): 087003. doi:10.1289/EHP3189.

Lei, Bingli et al. „Low-concentration BPF induced cell biological responses by the ERα and GPER1-mediated signaling pathways in MCF-7 breast cancer cells.” Ecotoxicology and Environmental Safety 165 (2018): 144-152. doi:10.1016/j.ecoenv.2018.08.102.

Okazaki, Hiroyuki et al. „Bisphenol AF as an Inducer of Estrogen Receptor β (ERβ): Evidence for Anti-estrogenic Effects at Higher Concentrations in Human Breast Cancer Cells.” Biological & Pharmaceutical Bulletin 40, 11 (2017): 1909-1916. doi:10.1248/bpb.b17-00427.

Okazaki, Hiroyuki et al. „Bisphenol AF as an activator of human estrogen receptor β1 (ERβ1) in breast cancer cell lines.” The Journal of Toxicological Sciences 43,5 (2018): 321-327. doi:10.2131/jts.43.321.

Kim, Ji-Youn et al. „Effects of bisphenol compounds on the growth and epithelial mesenchymal transition of MCF-7 CV human breast cancer cells.” Journal of Biomedical Research 31,4 (2017): 358-369. doi:10.7555/JBR.31.20160162.

Maffini, Maricel V et al. „Endocrine disruptors and reproductive health: the case of bisphenol-A.” Molecular and Cellular Endocrinology 254-255 (2006): 179-86. doi:10.1016/j.mce.2006.04.033.

Muñoz-de-Toro, Monica et al. „Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice.” Endocrinology 146,9 (2005): 4138-47. doi:10.1210/en.2005-0340.

Vandenberg, Laura N et al. „Perinatal exposure to the xenoestrogen bisphenol-A induces mammary intraductal hyperplasias in adult CD-1 mice.” Reproductive Toxicology 26,3-4 (2008): 210-9. doi:10.1016/j.reprotox.2008.09.015.

Betancourt, Angela M et al. „Proteomic analysis in mammary glands of rat offspring exposed in utero to bisphenol A.” Journal of Proteomics 73,6 (2010): 1241-53. doi:10.1016/j.jprot.2010.02.020.

Murray, Tessa J et al. „Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure.” Reproductive Toxicology 23,3 (2007): 383-90. doi:10.1016/j.reprotox.2006.10.002.

Acevedo, Nicole et al. „Perinatally administered bisphenol a as a potential mammary gland carcinogen in rats.” Environmental Health Perspectives 121,9 (2013): 1040-6. doi:10.1289/ehp.1306734.

Wadia, Perinaaz R et al. „Perinatal bisphenol A exposure increases estrogen sensitivity of the mammary gland in diverse mouse strains.” Environmental Health Perspectives 115, 4 (2007): 592-8. doi:10.1289/ehp.9640.

Ayyanan, Ayyakkannu et al. „Perinatal exposure to bisphenol a increases adult mammary gland progesterone response and cell number.” Molecular Endocrinology 25, 11 (2011): 1915-23. doi:10.1210/me.2011-1129.

Tucker, Deirdre K et al. „Evaluation of Prenatal Exposure to Bisphenol Analogues on Development and Long-Term Health of the Mammary Gland in Female Mice.” Environmental Health Perspectives 126, 8 (2018): 087003. doi:10.1289/EHP3189.

Rudel, Ruthann A et al. „Food packaging and bisphenol A and bis(2-ethyhexyl) phthalate exposure: findings from a dietary intervention.” Environmental Health Perspectives 119,7 (2011): 914-20. doi:10.1289/ehp.1003170.

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